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GIP(7-30)NH 2 was found to be a GIP-specific receptor antagonist that inhibits GIP-stimulated cAMP production and insulin release in a concentration-dependent manner

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We are also interested in the correlated changes across all three modalities, as the comparison between brain metabolism and function has important implications for medical imaging biomarkers, both when metabolism and function are correlated [27] and also when they diverge [28]